BACKGROUND: The COVID-19 mortality rate amongst kidney transplant recipients (KTR) is 24%,(1) 7 times higher than in the general population. Because of this, transplant recipients were given priority status for vaccination in Australia. However, vaccine efficacy in this group is questioned due to the use immunosuppressive medications. AIM: To compare COVID-19 vaccine (BNT162b2 and ChAdOx1) immunogenicity in KTRs with that of their cohabitants, with a focus on correlates of protection from infection,(2) in order to inform policy for the roll-out of booster vaccines. RESULTS/DISCUSSION: 43% of KTRs [12/28] produced anti-SARS-CoV-2 spike IgG after 2 vaccine doses (compared with 100% of cohabitants [32/32]). However, the median antibody titre for KTRs who did seroconvert was 151-fold lower than that of their cohabitants. Concordantly, KTRs had a heavily reduced capacity for virus neutralisation; even patients with promising antibody titres failed to effectively inhibit viral entry into ACE2+ cells. With respect to cellular immunity, we found that KTRs had, on average, 10-fold fewer spike-reactive IFNg-secreting cells following 2 vaccine doses, with the exception of patients receiving rapamycin. KTRs taking rapamycin (n=4) had higher antibody titres and a 28-fold greater frequency of spike-reactive IFNg-secreting cells compared with KTRs receiving standard of care triple therapy. Concordantly, activation-induced marker expression revealed KTRs receiving rapamycin to have significantly higher frequencies of spike-reactive CD4+ (4-1BB+OX40+) and CD8+ (4-1BB+CD69+) T cells compared with healthy cohabitants and other KTRs. CONCLUSION: KTRs display poor humoral and cellular immunity to the COVID-19 vaccines that are available in Australia. By contrast, their cohabitants demonstrate robust immune correlates of protection from infection and transmission. We recommend priority vaccination of close household contacts of KTRs, and have initiated a randomised controlled trial to test modification of immunosuppression with rapamycin as an adjuvant therapy to improve vaccine responses in this vulnerable group.